Virology

[1] kshv orf59 and pan rna recruit histone demethylases to the viral chromatin during lytic reactivation

[2] kshv rta promoter specification and viral reactivation

[3] fus negatively regulates kaposi’s sarcoma-associated herpesvirus gene expression

[4] kshv targeted therapy: an update on inhibitors of viral lytic replication

[5] cell cycle regulatory functions of the kshv oncoprotein lana

[6] the ubiquitin system and kaposi’s sarcoma-associated herpesvirus

[7] dna tumor virus regulation of host dna methylation and its implications for immune evasion and oncogenesis

[8] DNA-Packing Portal and Capsid-Associated Tegument Complexes in the Tumor Herpesvirus KSHV.

[9] Kaposi Sarcoma Herpes Virus (KSHV) infection inhibits macrophage formation and survival by counteracting Macrophage Colony-Stimulating Factor (M-CSF)-induced increase of Reactive Oxygen Species (ROS), c-Jun N-terminal kinase (JNK) phosphorylation and autophagy

[10] Signatures of oral microbiome in HIV-infected individuals with oral Kaposi's sarcoma and cell-associated KSHV DNA.

[11] kshv orf59 and pan rna recruit histone demethylases to the viral chromatin during lytic reactivation

[12] kshv rta promoter specification and viral reactivation

[13] kshv targeted therapy: an update on inhibitors of viral lytic replication

[14] cell cycle regulatory functions of the kshv oncoprotein lana

[15] kshv reactivation and novel implications of protein isomerization on lytic switch control