neurochemistry international

journal of neuroscience research

[1] Intranasal wnt-3a alleviates neuronal apoptosis in early brain injury post subarachnoid hemorrhage via the regulation of wnt target PPAN mediated by the moonlighting role of aldolase C.

[2] urine test strips to exclude cerebral spinal fluid blood

[3] informed consent: the rate-limiting step in acute stroke trials

[4] diagnostic value of 64-slice cta in detection of intracranial aneurysm in patients with sah and comparison of the cta results with 2d-dsa and intraoperative findings

[5] medical image of the week: bronchial clot removal via cryotherapy

[6] tratamiento endovascular de 473 aneurismas intracraneanos: resultados angiográficos y clínicos. experiencia colombiana, 1996-2008

[7] Sulforaphane enhances the activity of the Nrf2–ARE pathway and attenuates inflammation in OxyHb-induced rat vascular smooth muscle cells

[8] The role of TLR4 and HO-1 in neuroinflammation after subarachnoid hemorrhage.

[9] Prioritization and Timing of Outcomes and Endpoints After Aneurysmal Subarachnoid Hemorrhage in Clinical Trials and Observational Studies: Proposal of a Multidisciplinary Research Group

[10] Common Data Elements for Radiological Imaging of Patients with Subarachnoid Hemorrhage: Proposal of a Multidisciplinary Research Group

[11] Common Data Elements for Unruptured Intracranial Aneurysms and Aneurysmal Subarachnoid Hemorrhage: Recommendations from the Working Group on Hospital Course and Acute Therapies—Proposal of a Multidisciplinary Research Group

[12] Subarachnoid hemorrhage pattern predicts acute cerebral blood flow response in the rat

[13] Possible involvement of caspase-independent pathway in neuronal death after subarachnoid hemorrhage in mice

[14] Intranasal wnt-3a alleviates neuronal apoptosis in early brain injury post subarachnoid hemorrhage via the regulation of wnt target PPAN mediated by the moonlighting role of aldolase C.

[15] Intracranial hypertension in patients with aneurysmal subarachnoid hemorrhage: a systematic review and meta-analysis.

[16] Letter to the Editor Regarding "Increasing Frailty Predicts Worse Outcomes and Increased Complications Following Angiogram-Negative Subarachnoid Hemorrhages".

[17] A methodological remark on in vivo models of experimental subarachnoid hemorrhage

[18] Reduced tyrosine hydroxylase-like immunoreactivity around cerebral arteries after experimental subarachnoid hemorrhage in rats

[19] invasive and noninvasive multimodal bedside monitoring in subarachnoid hemorrhage: a review of techniques and available data

[20] early brain injury: a common mechanism in subarachnoid hemorrhage and global cerebral ischemia

[21] the impact of experimental preconditioning using vascular endothelial growth factor in stroke and subarachnoid hemorrhage

[22] acute microvascular changes after subarachnoid hemorrhage and transient global cerebral ischemia

[23] brain injury after transient global cerebral ischemia and subarachnoid hemorrhage

[24] pseudo-subarachnoid hemorrhage sign

[25] delayed traumatic subarachnoid hemorrhage in a polytraumatized patient with disseminated intravascular coagulation

[26] effect of aneurysmal subarachnoid hemorrhage on word generation

[27] cerebral vasospasm with ischemia following a spontaneous spinal subarachnoid hemorrhage

[28] prognostic factors in subarachnoid hemorrhage of idiopathic or aneurysmal origin

[29] serum magnesium levels and clinical outcome of aneurysmal subarachnoid hemorrhage: a study in 60 patients

[30] cerebral vasospasm after subarachnoid hemorrhage and tadalafil-nimodipine hypothesis

[31] the correlation between cerebral blood flow measured by bedside xenon-ct and brain chemistry monitored by microdialysis in the acute phase following subarachnoid hemorrhage

[32] postcesarean subarachnoid hemorrhage as the initial presentation of cerebral venous thrombosis

[33] a late complication of postpartum hellp syndrome: subarachnoid hemorrhage developed at the postpartum fourth day

[34] unique contribution of haptoglobin and haptoglobin genotype in aneurysmal subarachnoid hemorrhage

[35] effect of continuous cisternal cerebrospinal fluid drainage for patients with thin subarachnoid hemorrhage

[36] therapeutic implications of estrogen for cerebral vasospasm and delayed cerebral ischemia induced by aneurysmal subarachnoid hemorrhage

[37] P03 Heme oxygenase 1 – a potential early prognostic marker for neurological outcome after aneurysmal subarachnoid hemorrhage

[38] The calcimimetic R-568 attenuates subarachnoid hemorrhage-induced vasospasm through PI3K/Akt/eNOS signaling pathway in the rat model