[1] Myc signaling via the ARF tumor suppressor regulates p53-dependent apoptosis and immortalization

[2] Bmi-1 collaborates with c-Myc in tumorigenesis by inhibiting c-Myc-induced apoptosis via INK4a/ARF

[3] Disruption of the ARF-Mdm2-p53 tumor suppressor pathway in Myc-induced lymphomagenesis

[4] Senescence bypass screen identifies TBX2, which represses Cdkn2a (p19(ARF)) and is amplified in a subset of human breast cancers

[5] Loss of p16Ink4a confers susceptibility to metastatic melanoma in mice

[6] The alternative product from the human CDKN2A locus, p14(ARF), participates in a regulatory feedback loop with p53 and MDM2

[7] Induction of ARF tumor suppressor gene expression and cell cycle arrest by transcription factor DMP1

[8] Cdkn2a encodes functional variation of p16INK4a but not p19ARF, which confers selection in mouse lung tumorigenesis

[9] INK4a/ARF mutations accelerate lymphomagenesis and promote chemoresistance by disabling p53