the journal of allergy and clinical immunology

[1] Human STAT5b mutation causes dysregulated human natural killer cell maturation and impaired lytic function.

[2] perforinopathy: a spectrum of human immune disease caused by defective perforin delivery or function

[3] cytotoxic proteins and therapeutic targets in severe cutaneous adverse reactions

[4] the frequency of a91v in the perforin gene and the effect of tumor necrosis factor-α promoter polymorphism on acquired hemophagocytic lymphohistiocytosis

[5] intrinsic contribution of perforin to nk-cell homeostasis during mouse cytomegalovirus infection

[6] [Cellular and molecular effects of the antidepressant hyperforin on brain cells: Review of the literature].

[7] perforinopathy: a spectrum of human immune disease caused by defective perforin delivery or function

[8] natural killer cells from patients with recombinase-activating gene and non-homologous end joining gene defects comprise a higher frequency of cd56bright nkg2a+++ cells, and yet display increased degranulation and higher perforin content

[9] the frequency of a91v in the perforin gene and the effect of tumor necrosis factor-α promoter polymorphism on acquired hemophagocytic lymphohistiocytosis

[10] corrigendum: natural killer cells from patients with recombinase-activating gene and non-homologous end joining gene defects comprise a higher frequency of cd56bright nkg2a+++ cells, and yet display increased degranulation and higher perforin content

[11] intrinsic contribution of perforin to nk-cell homeostasis during mouse cytomegalovirus infection