BioMed research international

Frontiers in immunology

european journal of immunology

clinical & translational immunology

[1] CD4 T Responses Other Than Th1 Type Are Preferentially Induced by Latency-Associated Antigens in the State of Latent Infection.

[2] Circulating gluten-specific, but not CMV-specific, CD39 regulatory T cells have an oligoclonal TCR repertoire.

[3] Adenosine Generated by Regulatory T Cells Induces CD8 T Cell Exhaustion in Gastric Cancer through A2aR Pathway.

[4] multivariate analysis using high definition flow cytometry reveals distinct t cell repertoires between the foetal-maternal interface and the peripheral blood

[5] a sensitive method for detecting peptide-specific cd4+ t cell responses in peripheral blood from patients with myasthenia gravis

[6] Divergent SARS-CoV-2-specific T and B cell responses in severe but not mild COVID-19 patients.

[7] dopaminergic stimulation of myeloid antigen-presenting cells attenuates signal transducer and activator of transcription 3-activation favouring the development of experimental autoimmune encephalomyelitis

[8] how do cd4+ t cells detect and eliminate tumor cells that either lack or express mhc class ii molecules?

[9] quantifying susceptibility of cd4+ stem memory t-cells to infection by laboratory adapted and clinical hiv-1 strains

[10] cd161+cd4+ t cells are enriched in the liver during chronic hepatitis and associated with co-secretion of il-22 and interferon-gamma

[11] Correction: An “Escape Clock” for Estimating the Turnover of SIV DNA in Resting CD4+ T Cells.

[12] SA-4-1BBL costimulation inhibits conversion of conventional CD4+ T cells into CD4+ FoxP3+ T regulatory cells by production of IFN-γ.

[13] Dysregulation in IL-6 receptors is associated with upregulated IL-17A related signaling in CD4+ T cells of children with autism

[14] CD4+ T cells clear virus but augment disease in mice infected with respiratory syncytial virus. Comparison with the effects of CD8+ T cells

[15] CD4+ T cells clear virus but augment disease in mice infected with respiratory syncytial virus. Comparison with the effects of CD8+ T cells.

[16] differential expression of <i>smad</i> genes and <i>s1pr1</i> on circulating cd4+ t cells in multiple sclerosis and crohn’s disease

[17] correction to: a ccr5+ memory subset within hiv-1-infected primary resting cd4+ t cells is permissive for replication-competent, latently infected viruses in vitro

[18] tlr2 expression in peripheral cd4+ t cells promotes th17 response and is associated with disease aggravation of hepatitis b virus-related acute-on-chronic liver failure

[19] global stability of hiv infection of cd4+ t cells and macrophages with ctl immune response and distributed delays

[20] ccr5 gene editing of resting cd4+ t cells by transient zfn expression from hiv envelope pseudotyped nonintegrating lentivirus confers hiv-1 resistance in humanized mice

[21] computational modeling of heterogeneity and function of cd4+ t cells

[22] contribution of cd4+ t cells to the early mechanisms of ischemia- reperfusion injury in a mouse model of acute renal failure

[23] mhc class ii-restricted epitopes containing an oxido-reductase activity prompt cd4+ t cells with apoptosis-inducing properties

[24] how do cd4+ t cells detect and eliminate tumor cells that either lack or express mhc class ii molecules?

[25] transcriptional modulation of human endogenous retroviruses in primary cd4+ t cells following vorinostat treatment

[26] cd161+cd4+ t cells are enriched in the liver during chronic hepatitis and associated with co-secretion of il-22 and interferon-gamma

[27] Antigen presentation by mouse CD4+ T cells involving acquired MHC class II:peptide complexes: another mechanism to limit clonal expansion?

[28] Galectin 7 leads to a relative reduction in CD4+ T cells, mediated by PD-1